Atopic Dermatitis

ICD-11: EA80

Disease Overview

Atopic dermatitis (eczema) is a chronic inflammatory skin disease characterized by pruritus, erythema, and barrier dysfunction. It is a component of the atopic march (often preceding asthma and allergic rhinitis). Th2 skewing, filaggrin (FLG) deficiency, and epithelial barrier defects are central. Heritability ~75%; SNP-based h² ~0.15. Key genes include FLG (loss-of-function), IL4/IL13 (Th2), and TSLP. Allergens, irritants, climate, and stress trigger flares; microbiome and diet may modify risk.

Often begins in infancy/childhood; can persist or onset in adolescence. Psychosocial burden and transition to adult care are relevant.

Genetic Architecture Summary

GeneVariantGWAS pEvidenceStrength
FLGloss-of-function1.0e-50Filaggrin; barrier protein; null alleles strongly increase risk0.95
IL13rs205411.0e-20Th2 cytokine; type 2 inflammation0.85

Heritability

h² SNP: 0.15 Eczema GWAS (2020)

PRS notes: FLG status and PRS inform severity; transferability across ancestries variable.

Exposure Modifier Panel

ExposureDirectionStrengthConfidenceMechanism hypothesis
air-pollutionamplify0.6MEDIUM
psychosocial-stressamplify0.7HIGH
endotoxinbuffer0.5LOWEarly-life microbial exposure may protect

Population Equity Notes

GWAS ancestry breakdown: European-dominated; FLG effects vary by ancestry.

Transferability notes: FLG and IL13 replicate; PRS performance in non-European populations limited.

Data gaps: Multi-ancestry GWAS; G×E for allergens and microbiome.

Tissue Context

skin0.98
epidermis0.95

Visualizations

Risk Shift by Exposure Stratum

Population-level data only — does not predict individual risk

Tissue Relevance

References

  1. 1.Paternoster L, et al. (2015). Multi-ancestry genome-wide association study of 21,000 cases and 95,000 controls identifies new risk loci for atopic dermatitis. Nature Genetics. doi:10.1038/ng.3424
  2. 2.Kantor R, Silverberg JI (2016). Environmental and lifestyle factors in atopic dermatitis. Clinical Reviews in Allergy & Immunology. doi:10.1007/s12016-016-8548-5