Bipolar Disorder

ICD-11: 6A60

Disease Overview

Bipolar disorder is a mood disorder characterized by recurrent episodes of mania or hypomania and depression. Pathophysiology involves dysregulation of monoaminergic and glutamatergic systems, circadian rhythm disruption, and neuroplasticity deficits. Heritability is high (~70–80%); SNP-based h² ~0.25. Key loci include CACNA1C (calcium signaling), ANK3 (ankyrin-G), and genes in dopamine and circadian pathways. Stress, sleep deprivation, and substance use are environmental triggers. Adolescence and early adulthood are peak onset periods.

Onset peaks in late adolescence (15–24). Sleep disruption and substance use in this window amplify risk. Early intervention improves long-term outcomes.

Genetic Architecture Summary

GeneVariantGWAS pEvidenceStrength
CACNA1Crs10067371.0e-8Voltage-gated calcium channel; neuronal excitability and mood regulation0.82
ANK3rs109943365.0e-9Ankyrin-G; axon initial segment; neuronal firing0.78

Heritability

h² SNP: 0.25 PGC bipolar GWAS (2019)

PRS notes: PRS shows modest discrimination; clinical utility under study.

Exposure Modifier Panel

ExposureDirectionStrengthConfidenceMechanism hypothesis
psychosocial-stressamplify0.85HIGHStress triggers episodes; HPA axis and circadian disruption
alcoholamplify0.7MEDIUM

Population Equity Notes

GWAS ancestry breakdown: Discovery predominantly European.

Transferability notes: Multi-ancestry efforts ongoing.

Data gaps: Diverse cohorts; G×E in bipolar disorder.

Tissue Context

prefrontal cortex0.90
hippocampus0.85

Visualizations

Risk Shift by Exposure Stratum

Population-level data only — does not predict individual risk

Tissue Relevance

References

  1. 1.Stahl EA, et al. (2019). Genome-wide association study of bipolar disorder. Nature Genetics. doi:10.1038/s41588-019-0397-8
  2. 2.Rowland TA, Marwaha S (2018). Environmental risk factors for bipolar disorder. British Journal of Psychiatry. doi:10.1192/bjp.2018.132